Estrogenic endocrine disruptors and autoimmune disease

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منابع مشابه

Estrogenic endocrine disruptors and autoimmune disease.

min C in the treatment of shingles: results of a multicenter prospective cohort study. Med Sci Monit 2012;18:CR215–24. 6. Kim HJ, Lee YH, Im SA, Kim K, Lee CK. Cyclooxygenase inhibitors, aspirin and ibuprofen, inhibit MHC-restricted antigen presentation in dendritic cells. Immune Netw 2010;10:92–98. 7. Paccani SR, Boncristiano M, Ulivieri C, D’Elios MM, Del Prete G, Baldari CT. Nonsteroidal ant...

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Endocrine disruptors and estrogenic effects on male reproductive axis.

Endocrine disruptors (e.g., polychlorinated biphenyls [PCBs], dichlorodiphenyl-trichloroethane [DDT], dioxin, and some pesticides) are estrogen-like and anti-androgenic chemicals in the environment. They mimic natural hormones, inhibit the action of hormones, or alter the normal regulatory function of the endocrine system and have potential hazardous effects on male reproductive axis causing in...

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Endocrine Disruptors: Human Disease and Disease Models

Breast cancer strikes one out of eight women in Switzerland. The increase in breast cancer incidence over the past 70 years parallels an enormous increase of man-made, persistent chemicals in our environment; some of which have endocrine-disrupting properties in wildlife and/or in animal models. Epidemiological evidence is strong that a woman’s risk to get breast cancer is linked to her reprodu...

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Deleterious effects of estrogenic endocrine disruptors on marine organisms: Histological Observed Effects and Some Novel Useful Monitoring Bioassays

Aquatic environments receive significant levels of chemical contaminants generated by human activities. Among these pollutants, we noticed the xenobiotics known as reproductive toxicants and endocrine disruptors. The endocrine disruption in wildlife has been the subject of many reviews and workshops in recent years. Field observations of reproductively abnormal organisms and population declines...

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Endocrine Disruptors

Law and science combine in the estimation of risks from endocrine disruptors (EDs) and actions for their regulation. For both, dose–response models are the causal link between exposure and probability (or percentage change) of adverse response. The evidence that leads to either regulations or judicial decrees is affected by uncertainty and limited knowledge, raising difficult policy issues that...

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ژورنال

عنوان ژورنال: International Journal of Epidemiology

سال: 2014

ISSN: 0300-5771,1464-3685

DOI: 10.1093/ije/dyu133